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Tesamorelin

Established

Also known as Egrifta, Egrifta SV

GHRH analog

An FDA-approved GHRH analog that targets visceral fat.

References
1
Human-grade claims
2 of 2
Evidence floor
Established
Status
Approved

A stabilized growth-hormone-releasing-hormone analog that raises the body's own GH pulses. It is FDA-approved to reduce excess visceral abdominal fat in adults with HIV-associated lipodystrophy, backed by controlled trials — the best-evidenced GH-axis peptide here.

GH-axisGHRHapproved

How it works

Tesamorelin is a protected copy of GHRH, the hormone that tells the pituitary to release growth hormone. An added acyl cap on the N-terminus shields it from rapid breakdown, so it drives natural GH pulses rather than replacing GH directly. More GH in turn mobilizes visceral fat.

Sequence: Human GHRH(1-44) with an N-terminal trans-3-hexenoyl cap and C-terminal amideThe acyl cap protects against DPP-4 cleavage and is the load-bearing structural feature.

What it's studied for

Safety & limitations

The label warns of glucose intolerance and diabetes, injection-site reactions, and possible fluid retention; it is contraindicated in active malignancy and pregnancy. Visceral-fat benefit reverses if the drug is stopped.

[1] FDA label — Egrifta SV (accessdata)

Regulatory & legal status

Approved

FDA-approved (2010) to reduce excess visceral abdominal fat in adults with HIV-associated lipodystrophy. Marketed as Egrifta / Egrifta SV.

[1] FDA label — Egrifta SV (accessdata)

Frequently asked

Is tesamorelin a growth hormone?
No — it prompts your own pituitary to release growth hormone, rather than being GH itself. That is why it is described as a GHRH analog.

References

Written by Twenty Residues editorial. Reviewed by Pending named medical review.

Last updated September 19, 2026.

Update history
  • 2026-09-19 — Sourced review — every claim checked against primary literature; sequence, regulatory status, safety, and FAQs added.